Interested in how this goes. I'm biased to have simplicity - just linoleic acid - to win by 90% or more here.
Also, it seems that a keto diet with moderate amounts of pufa(evoo, eggs, avocado, fish, occasional nuts and chicken...) doesn't accumulate much pufa in the fat cells, especially if you're already lean(Nick Norwitz, Raobb Wolf or Peter Dobromylskiy in his early keto years). Pufas are treated by the body more or less like mcts. Chucked into the ketone furnace. Obesity probably changes things. Most importantly, such a diet doesn't deplete linoleic acid already in the fat cells as efficiently.
Of course, how does one be “already lean?” This might just be genetic selection bias heh. I haven’t been “lean” since puberty.
I don’t think obesity changes this; I think obesity is created from PUFAs in most people because they are not hyper PUFA detoxers. Maybe these naturally lean people are.
"Already lean", as in someone who either already depleted their linoleic acid or was never overweight to begin with. Neither of them were doing keto in childhood, but both could likely follow such a version of keto(with evoo, eggs, avocado, and occasional bacon/drumsticks) and not have those pufas get stored in fat cells. In other words, I think the bar is lower when fat cell pufa is not an issue.
What obesity might possibly change in that equation is the much larger absolute amount of insulin sensitive total fat mass. This has a significant effect on fasting insulin and other hormones by extension. Lower fat burning at rest, changes in appetite, fuel partitioning... It can certainly complicate things. If I settle on keto after my depletion is done, I probably won't worry about those occasional excursions.
Concur, and interested to see the results! Would also be interested to see what happens if you just say bollocks to it and eat random no-PUFA stuff for a bit according to taste.
Sign and signal are both from Latin signum (a sign) I think, further back it's probably related to ancient words for following or cutting. See also sequel, section, etc.
I have tracked body temp in the past, but it's always just a steady 98.6°F. I don't think I ever had low body temp.
I do periodic OQCs and presumably the linoleic acid on those would go down over time. Unfortunately, so far on my timescale (~2 years of testing?) it's not exactly a steady linear drop. It goes so slow that it might take 6-18 months to see a signal in the noise.
Interested in how this goes. I'm biased to have simplicity - just linoleic acid - to win by 90% or more here.
Also, it seems that a keto diet with moderate amounts of pufa(evoo, eggs, avocado, fish, occasional nuts and chicken...) doesn't accumulate much pufa in the fat cells, especially if you're already lean(Nick Norwitz, Raobb Wolf or Peter Dobromylskiy in his early keto years). Pufas are treated by the body more or less like mcts. Chucked into the ketone furnace. Obesity probably changes things. Most importantly, such a diet doesn't deplete linoleic acid already in the fat cells as efficiently.
Of course, how does one be “already lean?” This might just be genetic selection bias heh. I haven’t been “lean” since puberty.
I don’t think obesity changes this; I think obesity is created from PUFAs in most people because they are not hyper PUFA detoxers. Maybe these naturally lean people are.
"Already lean", as in someone who either already depleted their linoleic acid or was never overweight to begin with. Neither of them were doing keto in childhood, but both could likely follow such a version of keto(with evoo, eggs, avocado, and occasional bacon/drumsticks) and not have those pufas get stored in fat cells. In other words, I think the bar is lower when fat cell pufa is not an issue.
What obesity might possibly change in that equation is the much larger absolute amount of insulin sensitive total fat mass. This has a significant effect on fasting insulin and other hormones by extension. Lower fat burning at rest, changes in appetite, fuel partitioning... It can certainly complicate things. If I settle on keto after my depletion is done, I probably won't worry about those occasional excursions.
Concur, and interested to see the results! Would also be interested to see what happens if you just say bollocks to it and eat random no-PUFA stuff for a bit according to taste.
Sign and signal are both from Latin signum (a sign) I think, further back it's probably related to ancient words for following or cutting. See also sequel, section, etc.
The issue is the PUFA in your mitochondrial cell membrane, right? So a steep cut to reduce adipose wouldn’t speed the process?
If you know how to do a steep cut that leads to sustainable fat loss, please let me know :)
What would linoleum acid depletion look like on periodic Omega Quant testing?
Do you try to track body temp?
I have tracked body temp in the past, but it's always just a steady 98.6°F. I don't think I ever had low body temp.
I do periodic OQCs and presumably the linoleic acid on those would go down over time. Unfortunately, so far on my timescale (~2 years of testing?) it's not exactly a steady linear drop. It goes so slow that it might take 6-18 months to see a signal in the noise.